Preliminary reports reveal conolidine may perhaps inhibit certain ion channels, lessening neuronal excitability and limiting ache indicators. This system is particularly pertinent in neuropathic pain, in which abnormal signaling results in persistent pain. Furthermore, conolidine appears to affect G protein-coupled receptor (GPCR) pathways integral to suffering notion. Computational d... https://juliom123tiu0.wikihearsay.com/user